Spring Break
Form of bacteria led to student death
PEARMAN PARKER
Issue date: 12/4/07 Section: News
- Page 1 of 1
An autopsy report released Monday stated the cause of death for Michael Hendrickson was not bacterial meningitis, according to the county coroner. Hendrickson, a University student, died Saturday.
"The cause of death was Neisseria meningitidis," said Bobby Tribble, coroner for Athens-Clarke County.
The Health Center has seen 30 students and treated 25 with antibiotics, said Liz Rachun, public relations coordinator for the Health Center.
Rachun said St. Mary's Hospital gave 16 people antibiotics, including eight staff members. One person received treatment in Augusta, and 11 people received antibiotics at other locations.
"In total, 53 people received prophylaxis (antibiotics)," Rachun said. "We received 45 calls on the hotline so far."
Students can contact the Health Center hotline at 706-542-8608 with concerns.
"The cause of death was Neisseria meningitidis," said Bobby Tribble, coroner for Athens-Clarke County.
The Health Center has seen 30 students and treated 25 with antibiotics, said Liz Rachun, public relations coordinator for the Health Center.
Rachun said St. Mary's Hospital gave 16 people antibiotics, including eight staff members. One person received treatment in Augusta, and 11 people received antibiotics at other locations.
"In total, 53 people received prophylaxis (antibiotics)," Rachun said. "We received 45 calls on the hotline so far."
Students can contact the Health Center hotline at 706-542-8608 with concerns.
Viewing Comments 1 - 2 of 2
Joseph Scott
posted 12/04/07 @ 10:28 AM EST
In your article about the cause of death of Michael Hendrickson, you mentioned that the cause of death was not due to bacterial meningitis. Then you go on to say that the cause of death due to infection by the bacteria that causes meningitis, Neisseria meningitidis. (Continued…)
mcgdawg
mcgdawg
posted 12/05/07 @ 1:01 AM EST
Joseph
The bacterium Neisseria meningitidis does in fact cause bacterial meningitis. This occurs when the bacteria enters the cerebrospinal fluid, giving it access to the meninges of the brain. (Continued…)
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